Mercury expert Professor Boyd Haley is concerned that the huge
increase in autism may be due to exposure to mercury from mothers’
amalgam fillings while in the womb. He also believes that an interaction
between the mercury-based vaccine preservative thimerosal and
the male hormone testosterone may play a part. His testimony before
the US House Government Reform Committee (14.11.02) included
the following:
Mercury in the mouth and intestines can mutate into more
toxic organic mercury compounds like methyl-mercury, facilitating
its uptake by babies in the womb [1,2,3]
Methylthiol, one of the major neurotoxins produced during
gum diseases, reacts immediately with the mercury ion Hg2+,
creating extremely dangerous organic mercury-thiol compounds.
Like methyl-mercury, these can easily penetrate the blood/bowel
and blood-brain barriers of adults,children and foetuses.
This latter might explain why gum disease in the mother
is the main risk factor for pre-term low birth weight babies
Raised mercury levels in mothers' fluids have been linked
with even higher levels present in the cord blood and in
the meconium (faeces) passed by the baby during the first
days after birth [4]
Levels of mercury in the ‘birth hair’ of autistic
children are usually lower than in normal children, whilst
their blood levels of mercury are higher. In normal children,
the level of mercury in ‘birth hair’ increases
in line with the number of amalgam fillings in the mother’s
mouth. In autistic children there is very little excretion
of mercury into the ‘birth hair’ no matter how
many amalgam fillings their mother has. When children with
autism are exposed to mercury in tests, they retain higher
amounts in the body than normal children. This all suggests
that the bodies of children with autism are less effective
at excreting mercury [5]
The mercury-based vaccine preservative thimerosal kills
neurons, particularly when inorganic mercury, aluminium,
lead or cadmium are present
Whereas the female hormone oestrogen decreases thimerosal’s
toxic effects, the male hormone testosterone greatly increases
its toxicity. Exposing neurons to even a tiny concentration
of either thimerosal or testosterone alone killed 5% in
three hours. Three hours exposure to the same concentration
of thimerosal with just one nanomolar of testosterone added
killed all 100%. This may explain the four-to-one
ratio of boys to girls that become autistic and the fact
that boys represent the vast majority of the severe cases
of autism
Some children are born autistic. Others develop autism,
possibly due to exposure to mercury in early childhood from
vaccines, food or pollution. In a normal child, it is quite
plausible that protective compounds in the body such as
glutathione and metallothionine (which bind with mercury
and other heavy metals), would prevent neurological damage.
However, when a foetus is exposed to mercury its stores
of these compounds are reduced
These compounds also decrease in the body with age, disease,
other toxic exposures, oxidative stress and genetic susceptibility,
possibly explaining why Parkinson’s and Alzheimer’s
have, until recently, been primarily diseases of older people
N.B. It is the mercury retained in the body’s cells that
causes damage, not the mercury which has been excreted in the
urine, hair and faeces.
MMR - a hypothesis
Our studies found that the MMR triple jab (which does not contain
thimerosal) was far less neurotoxic than any thimerosal-containing
vaccines. If, however, it was given to a child whose blood/bowel
or blood/brain barrier had already been damaged by exposure
to, say, mercury from the mother’s amalgam fillings or
an earlier jab containing thimerosal, the live measles virus
in particular could easily penetrate the gut or brain where
it could trigger substantial damage.
Ed.- (i) Recently published research found that children with
autism had significantly lower levels of glutathione, homocysteine
and other amino acids essential for protecting the body against
heavy metals like mercury. [6]
(ii) A copy of the US International Academy of Oral Medicine
and Toxicology’s list of ‘amalgam-free’ dentists
may be obtained from the Green Health Watch office. Please send
a large stamped addressed envelope. It can also be downloaded
from website: www.amalgam.ukgo.com/ukdent.htm
[1] Kingman et al. Journal of Dental
Research 1998;V77(3):461
[2] Mackert,JR & Bergland,A. Critical Revues in Oral Biology
and Medicine 1997;8(4):410-36 (states it would take 450-530
amalgam surfaces to produce 30 micrograms mercury/g creatinine
of urine mercury per day (roughly estimated as 0.067 to 0.057
mg/day/surface)
[3] Holmes,A et al. International Journal of Toxicology 2003;V22:4
[4] Ramirez,GB et al. Pediatrics 2000;V106(4):774-81
[5] see e.g., Razagui,IB et al. Biological Trace Element Research
2001;81(1):1-19
Drasch,G et al. European Journal of Pediatrics 1995;154(7):585-86
Lorscheider,FL et al. Neuroreport 2001;12(4):733-37
[6] Saxe et al. Journal of American Dental Association 1999;V130:191
[7] Frustaci et al.
Journal of the American College of Cardiology 1999;v33(6):1578-83
[8] see e.g., Lorscheider,FL et al. FASEB Journal 1995;9:504-08
[9] see e.g., Kingman,A et al.
Journal of Dental Research 1998;77(3):461-71
[10] Chew,CL et al. Clinical Preventive Dentistry 1991;13(3):5-7
[11] see e.g., Reinhardt,JW.
Advances in Dental Research 1992;6;110-113
[12] see e.g., Nylander,M et al.
Journal of Swedish Dentistry 1987;11:179-187 and 1989;13:235-43
Zander,D et al.
Zentralblatt fur Hygiene und Umweltmedizin 1992;192(5):447-54
[13] Haley,B. Nordisk Tidsskrift for Biologisk Medisin 2003
[14] see e.g., Opitz,H. Clinical Neuropathology 1996;15(3):130-44
[15] see e.g. Echeverria,D et al. FASEB Journal 1998;12(11):971-80
Aschner,M & Aschner,JL.
Neuroscience and Biobehavioral Reviews 1990;14(23):169-76
[16] see e.g. Hua,MS et al. Brain Injury 1996;10(5):377-84
Siblerud,RL. Psychological Reports 1992;70(3 Pt 2);1139-51
[17] see e.g. Heintze,U et al.
Scandinavian Journal of Dental Research 1983;9(2):150-52
[18] Heintze et al.
Scandinavian Journal of Dental Research 1983;V91:150
[19] Rowland et al. Experientia
1975;V31:1064
[20] Leistevuo,J et al. Caries Research 2001;V35(3):163
[21] see e.g. Siblerud,RL.
The Science of the Total Environment 1990;99(1-2):23-25
Zentralblatt fur Hygiene und Umweltmedizin 1996 Nov: 199)1):
69-75